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Health, Well-Being & Alternative Medicine
Need health? Go take a PEA
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<blockquote data-quote="OakFieldAlienz444" data-source="post: 253197" data-attributes="member: 12609"><p>[URL unfurl="true"]https://pubmed.ncbi.nlm.nih.gov/31396087/[/URL]</p><p></p><p></p><p>[URL unfurl="true"]https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9138306/[/URL]</p><p>Palmitoylethanolamide (PEA) stands out among endogenous lipid mediators for its neuroprotective, anti-inflammatory, and analgesic functions. PEA belonging to the N-acetylanolamine class of phospholipids was first isolated from soy lecithin, egg yolk, and peanut flour. It is currently used for the treatment of different types of neuropathic pain, such as fibromyalgia, osteoarthritis, carpal tunnel syndrome, and many other conditions. The properties of PEA, especially of its micronized or ultra-micronized forms maximizing bioavailability and efficacy, have sparked a series of innovative research to evaluate its possible application as therapeutic agent for neurodegenerative diseases. Neurodegenerative diseases are widespread throughout the world, and although they are numerous and different, they share common patterns of conditions that result from progressive damage to the brain areas involved in mobility, muscle coordination and strength, mood, and cognition. The present review is aimed at illustrating in vitro and in vivo research, as well as human studies, using PEA treatment, alone or in combination with other compounds, in the presence of neurodegeneration. Namely, attention has been paid to the effects of PEA in counteracting neuroinflammatory conditions and in slowing down the progression of diseases, such as Alzheimer’s disease, Parkinson’s disease, Huntington’s disease, Frontotemporal dementia, Amyotrophic Lateral Sclerosis, and Multiple Sclerosis. Literature research demonstrated the efficacy of PEA in addressing the damage typical of major neurodegenerative diseases.</p><p></p><p>[URL unfurl="true"]https://www.frontiersin.org/articles/10.3389/fphar.2021.748021/full[/URL]</p><p></p><p></p><p></p><p>PEA is in the cannibanoid family but is not a drug or psychoactive, it is produced in your own body and found in egg yolk and increases the effectiveness of any other analgesic or pain reliever! It can be purchased online at Amazon, Ebay and other retailers</p><p><a href="https://www.whria.com.au/wp-content/uploads/2016/10/PEA-for-nerve-pain-and-inflammation.pdf" target="_blank">PEA-for-nerve-pain-and-inflammation.pdf (whria.com.au)</a></p><p></p><p>The potential clinical applications of PEA are quite broad, but research and popular use have focused on its use as an anti-inflammatory and pain-relieving agent in conditions like low back pain, sciatica, osteoarthritis, etc. Preclinical and human studies have also investigated its effects on depression, boosting mental function and memory, autism, multiple sclerosis, obesity, and metabolic syndrome. While it shares many features comparable to cannabidiol (CBD), the advantage of PEA is that it has better science to support its use.</p></blockquote><p></p>
[QUOTE="OakFieldAlienz444, post: 253197, member: 12609"] [URL unfurl="true"]https://pubmed.ncbi.nlm.nih.gov/31396087/[/URL] [URL unfurl="true"]https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9138306/[/URL] Palmitoylethanolamide (PEA) stands out among endogenous lipid mediators for its neuroprotective, anti-inflammatory, and analgesic functions. PEA belonging to the N-acetylanolamine class of phospholipids was first isolated from soy lecithin, egg yolk, and peanut flour. It is currently used for the treatment of different types of neuropathic pain, such as fibromyalgia, osteoarthritis, carpal tunnel syndrome, and many other conditions. The properties of PEA, especially of its micronized or ultra-micronized forms maximizing bioavailability and efficacy, have sparked a series of innovative research to evaluate its possible application as therapeutic agent for neurodegenerative diseases. Neurodegenerative diseases are widespread throughout the world, and although they are numerous and different, they share common patterns of conditions that result from progressive damage to the brain areas involved in mobility, muscle coordination and strength, mood, and cognition. The present review is aimed at illustrating in vitro and in vivo research, as well as human studies, using PEA treatment, alone or in combination with other compounds, in the presence of neurodegeneration. Namely, attention has been paid to the effects of PEA in counteracting neuroinflammatory conditions and in slowing down the progression of diseases, such as Alzheimer’s disease, Parkinson’s disease, Huntington’s disease, Frontotemporal dementia, Amyotrophic Lateral Sclerosis, and Multiple Sclerosis. Literature research demonstrated the efficacy of PEA in addressing the damage typical of major neurodegenerative diseases. [URL unfurl="true"]https://www.frontiersin.org/articles/10.3389/fphar.2021.748021/full[/URL] PEA is in the cannibanoid family but is not a drug or psychoactive, it is produced in your own body and found in egg yolk and increases the effectiveness of any other analgesic or pain reliever! It can be purchased online at Amazon, Ebay and other retailers [URL='https://www.whria.com.au/wp-content/uploads/2016/10/PEA-for-nerve-pain-and-inflammation.pdf']PEA-for-nerve-pain-and-inflammation.pdf (whria.com.au)[/URL] The potential clinical applications of PEA are quite broad, but research and popular use have focused on its use as an anti-inflammatory and pain-relieving agent in conditions like low back pain, sciatica, osteoarthritis, etc. Preclinical and human studies have also investigated its effects on depression, boosting mental function and memory, autism, multiple sclerosis, obesity, and metabolic syndrome. While it shares many features comparable to cannabidiol (CBD), the advantage of PEA is that it has better science to support its use. [/QUOTE]
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